Wednesday, May 31, 2017

TOBACCO: THE SOURCE OF PHYSIOLOGICALLY ACTIVE COMPOUNDS

tobacco

When burned, tobacco in cigarettes, cigars, and pipe mixtures is the source of an array of physiologically active chemicals, many of which are closely linked to significant changes in normal body structure and function.  At the burning tip of the cigarette, the 900o C (1,652 F) heat oxidizes tobacco (as well as paper, wrapper, filter, and additives).  With each puff or smoke, the body is exposed to approximately 4,700 chemical compounds, hundreds of which are known to be physiologically active toxic, and carcinogenic.  The chemicals have their origin in the tobacco or the over 1,450 additives, including pesticides and other agricultural chemicals.  An annual 70,000 puffs taken in by the one-pack-a-day cigarette smoker results in an environment that makes the most polluted urban environment seem clean by comparison.
Particulate Phase
Cigarette, cigar and pipe smoke can be described on the basis of two phases.  These phases include a particulate phase and a gaseous phase.  The particulate phase includes nicotine, water and a variety of powerful chemicals known collectively as tar.  Tar includes phenol, cresol, pyrene, DDT, and a benzene-ring group of compounds that includes benzo [a] pyrene.  Most of the carcinogenic compounds are found within the tar.  A person who smokes one pack of cigarettes per day will collect four ounces of tar in his or her lungs in a year.  Only the gases and the smallest particles reach the small sacs of the lungs, called the alveoli, where oxygen exchange occurs.  The carcinogen-rich particles from the particulate phase are deposited somewhere along the air passage leading to the lungs.
Gaseous Phase
The gaseous phase of tobacco smoke, like the particulate phase, is composed of a variety of physiologically active compounds, including carbon monoxide, carbon dioxide, ammonia, hydrogen cyanide, isopyrene, acetaldehyde, and acetone.  At least sixty of these compounds have been determined to be carcinogens or co-carcinogenic promoters, thus capable of stimulating the development of cancer.  Carbon monoxide is, however, the most damaging compound found in this component of tobacco smoke.
Carbon Monoxide
Like every inefficient engine, a cigarette, cigar, or pipe burns (oxidizes) its fuel with less than complete conversion into carbon dioxide, water, and heat.  As a result of this incomplete oxidation, burning tobacco forms carbon monoxide (CO) gas.  Carbon monoxide is one of the most harmful components of tobacco smoke.
Carbon monoxide is a colorless, odorless, tasteless gas that possess a very strong physiological attraction for hemoglobin, the oxygen-carrying pigment on each red blood cell.  When CO is inhaled, it quickly bonds with hemoglobin and forms a new compound, carboxyhemoglobin.  In this form, hemoglobin is unable to transport oxygen to the tissue and cells where it is needed.
Although it is true that normal body metabolism always keeps an irreducible minimum of CO in our blood (0.5% to 1%), the blood of smokers may have levels of 5% to 10% CO saturation.  We are exposed to additional CO from environmental sources such as automobiles and buses and other combustion of fossils fuels.  By combining a smoker’s CO with environmental CO, it is little wonder that smokers more easily become out of breath than nonsmokers.  The half-life of CO combined with hemoglobin is approximately 4 to 6 hours.  Most smokers replenish their level of CO saturation at far shorter intervals than this.
As mentioned, the presence of excessive levels of carboxyhemoglobin in the blood of smoker leads to shortness of breath and lowered endurance.  Because an adequate oxygen supply to all body tissues is critical for normal functioning, any oxygen reduction can have a serious impact on health.  Brain function may be eventually reduced, reactions and judgment are dulled, and of cource, cardiovascular function is impaired.  Fetuses are especially at risk for this oxygen deprivation (hypoxia) because fetal development is so critically dependent on a sufficient oxygen supply from the mother. 
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